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primary transformant DNA with a human Alusequence and a camr. These results should be interpreted with caution. The question is whether these apparently H. pylori-negative subjects were indeed truly negative or they eventually led to loss of the infection due to sever gastric atrophy. Because, in our study, gastric atrophy and H. pylori infection were based on serological diagnosis and the diagnosis happened to be imperfect, we might not discriminate severe atrophy exactly and further biological studies with histological assessment are needed to confirm the association. The functional changes caused by Gab1 polymorphism are not known and may be linkage disequilibrium with another gene. These are deficiencies of this study but we hope that our epidemiological and biologically plausible observation would stimulate interest in the study of the molecular mechanisms of action of this polymorphism. Because these results were based on the low frequency of A/A, they might be inconsistent due to the random errors. Studies of a larger size are needed to confirm our finding. This study, however, had 80% power to detect an absolute difference in the frequency of G/A+A/A, given 44% in the case and 56% in the control.. Among patients with H. pylori, the difference between the severity of gastritis in those with anemia and those without anemia was not statistically significant (p = 0.382) and no correlation was found between degree of H. pylori colonization and anemia.. and/or enzymatic level where can i purchase prednisone which could be responsible for the change in.
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